Please use this identifier to cite or link to this item: http://repo.tma.uz/xmlui/handle/1/2635
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dc.contributor.authorJavohir Mullokulov., Khalmurad Akhmedov
dc.date.accessioned2025-12-15T05:02:28Z
dc.date.available2025-12-15T05:02:28Z
dc.date.issued2025
dc.identifier.citationToshkenten_US
dc.identifier.urihttp://repo.tma.uz/xmlui/handle/1/2635
dc.description.abstractReactive Arthritis (ReA) is an immune-mediated form of spondyloarthritis, often triggered by genitourinary or gastrointestinal infections. Current therapeutic strategies rely mainly on NSAIDs and cs-DMARDs, with biologics reserved for refractory disease. However, limited progress has been made in exploring novel targeted agents. Iguratimod 25 mg, an oral immunomodulatory drug approved for rheumatoid arthritis in Asia, has demonstrated anti-inflammatory and cytokine-suppressive properties, but evidence in ReA is scarceen_US
dc.language.isoenen_US
dc.subjectTo evaluate the clinical efficacy and cytokine-modulating effects of Iguratimod 25 mg in patients with Reactiveen_US
dc.titleNOVEL TREATMENT APPROACHES IN REACTIVE ARTHRITIS: CLINICAL AND CYTOKINE RESPONSES TO IGURATIMOD 25 MG COMPARED WITH BIOLOGIC AND CONVENTIONAL THERAPIESen_US
dc.typeThesisen_US
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