Please use this identifier to cite or link to this item: http://repo.tma.uz/xmlui/handle/1/362
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dc.contributor.authorAbduraxmanova N.M., Axm edov X.S., Norbo'toyev O.M.-
dc.date.accessioned2024-12-16T18:53:51Z-
dc.date.available2024-12-16T18:53:51Z-
dc.date.issued2024-
dc.identifier.citationToshkenten_US
dc.identifier.issn2181-7812-
dc.identifier.urihttp://repo.tma.uz/xmlui/handle/1/362-
dc.description.abstractRevmatoid artrit biriktiruvchi to'qimaning surunkali autoimmun tizimli yallig'lanish kassalligi b o ’lib, asosan b o’g ’imlarni eroziv-destruktiv progressirlanib boruvchi poliartrtit tipida zararlaydi. Revmatoid artrit patogenezi asosidagenetikdeterminarlangan autoimmun jarayonlar yotadi, bu jarayonlarning kelib chiqishiga limfotsitlarn- ing T-supressor funktsiyasi defitsiti moyillik qiladi. Maz- kur kasallik negizidayuzaga kelayotgan b o ’g ’im struktur- asining o'zgarishi - ankiloz, deformasiya va kontraktura shakllanishi ko'p darajada erta boshlangan davo bilan bog'liq.en_US
dc.language.isootheren_US
dc.relation.ispartofseriesUO'K;0009-0002-7978-0355-
dc.subjectrevmatoid artrit, revmatoid omil, nodif- ferensirlangan artrit.en_US
dc.titleRevmatoid artritning klinik-patogenetik xususiyatlarien_US
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