Exchange of iron in the patents with diffuse toxic goiter accompanied bu iron deficiency anemia

Abstract

Anemia has been an actual problem in the world for a long time because its etiology and pathogenesis have not been studied well enough. One of the etiologic factors of anemia is pathology of thyroid gland. Anemia in iodine deficiency has been researched well while studies on conditions leading to anemia in goiter patients are fragmentary. We have studied causes and results of anemia in diffuse toxic goiter. In diffuse toxic goiter with intensive hyperactivity thyroid gland there were detected changes in peripheral blood in the form of anemia, lymphocytosis, eosinophilia, hypogranulocytosis, and thrombocytopenia (U.E. Vinogradov, A.P.Shinkarkin, A.M. Poverenniy, 2003). We decided to research pathogenetic aspects of anemia, exchange of iron in diffuse toxic goiter. Iron deficiency (sideropenia or hypoferremia) is one of the most common of the nutritional deficiencies. Iron is present in all cells in the human body, and has several vital functions. All cells of human body have iron. This iron has several (certain) vital function. Too little iron can negatively affect these vital functions and lead to morbidity and death.