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Please use this identifier to cite or link to this item: http://repo.tma.uz/xmlui/handle/1/5107
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dc.contributor.authorAsal Abduvakhitova1, 2, Durdona Saipova2.-
dc.date.accessioned2026-09-05T13:21:00Z-
dc.date.available2026-09-05T13:21:00Z-
dc.date.issued2025-04-09-
dc.identifier.urihttp://repo.tma.uz/xmlui/handle/1/5107-
dc.description.abstractPulmonary hypertension (PH) is a significant complication in ESRD patients on HD, exacerbated by AV fistulas and involving dysregulation of endothelin-1 (ET-1) and nitric oxide (NO). Aim to evaluate the role of endothelin-1 (ET-1) and nitric oxide (NO) in the development and progression of PH in patients on program hemodialysis and with AV access.en_US
dc.language.isoen_USen_US
dc.publisherAmerican Journal of Kidney Diseasesen_US
dc.titleG-286. FEATURES OF THE MECHANISM OF DEVELOPMENT OF PULMONARY HYPERTENSION IN PATIENTS UNDERGOING HEMODIALYSIS THROUGH ARTERIOVENOUS ACCESS:en_US
dc.typeThesisen_US
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