Please use this identifier to cite or link to this item: http://repo.tma.uz/xmlui/handle/1/5216
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dc.contributor.authorT.Sh. Abdikadirova., A.V. Alieva., R.H. Trigulova., H.K. Nasirova-
dc.date.accessioned2026-09-16T11:46:57Z-
dc.date.available2026-09-16T11:46:57Z-
dc.date.issued2026-05-11-
dc.identifier.urihttp://repo.tma.uz/xmlui/handle/1/5216-
dc.description.abstractThe use of amiodarone in the treatment of cardiac arrhythmias is associated with a significant risk of thyroid dysfunction, particularly in patients living in iodine- deficient regions [1,2,6]. Excess iodine load, combined with compensatory instability of the thyroid gland, may lead to the development of thyrotoxicosis [1,5,6], which adversely affects the course of cardiovascular diseases and worsens prognosis.en_US
dc.language.isoen_USen_US
dc.publisherGermaniya (INTERNATIONAL CONFERENCE ON HEALTH & TECHNOLOGY) journalen_US
dc.subjectamiodarone, thyroid dysfunction, thyrotoxicosis, heart rate variability, iodine deficiency, arrhythmiasen_US
dc.titleTHYROID DYSFUNCTION ASSOCIATED WITH ANTIARRHYTHMIC THERAPY IN PATIENTS FROM IODINE-DEFICIENT REGIONS OF THE ARAL SEA AREAen_US
dc.typeArticleen_US
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