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dc.contributor.authorAzimova Sevara Bahodirovna1, Samieva Gulnoza Utkurovna 2, Khidirova Sojida Khusniddin Kizi 3,*-
dc.date.accessioned2025-01-13T09:28:27Z-
dc.date.available2025-01-13T09:28:27Z-
dc.date.issued2024-
dc.identifier.urihttp://repo.tma.uz/xmlui/handle/1/855-
dc.description.abstractLactase deficiency (LY) is a disorder of the functioning of the congenital or acquired digestive system that is accompanied by a violation of the absorption of lactose (milk shikari), especially in the small intestine. In the Normal state, milk sugar is broken down into monosaccharides glucose and galactose by the action of the enzyme lactase in the small intestine cavity and is actively transported in the small intestine. Clinical manifestations of enzyme deficiency are determined by lactase deficiency. The enzyme mainly activates in the vortices of the apical part of the enterocytes of the small intestine. Low enzyme activity increases the risk of developing malabsorption syndrome. Patalogy, which is common all over the world today, is a disease in which a violation of the digestion and absorption process in the intestines accompanies lactase deficiency. The relevance of studying this disease is that the young organism that is, newborns feed mainly on breast milk and dairy products and adapt to the external environment, in which the role of lactase is played. The article provides a brief overview of enzyme deficient, its ethylogical factors, and novel treatments.en_US
dc.language.isoen_USen_US
dc.publisherAmerican Journal of Medicine and Medical Sciencesen_US
dc.subjectLactose, Lactase deficiency, Ethylogical factors, Genetic factors, Pathogenesisen_US
dc.titlePathogenetic Basis for the Development of Lactase Enzyme Deficiencyen_US
dc.typeArticleen_US
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