Abstract:
Anemia has been an actual problem in the world for a long time because its etiology and
pathogenesis have not been studied well enough. One of the etiologic factors of anemia is
pathology of thyroid gland. Anemia in iodine deficiency has been researched well while
studies on conditions leading to anemia in goiter patients are fragmentary. We have studied causes and results of anemia in diffuse toxic goiter. In diffuse toxic goiter with intensive
hyperactivity thyroid gland there were detected changes in peripheral blood in the form of
anemia, lymphocytosis, eosinophilia, hypogranulocytosis, and thrombocytopenia (U.E.
Vinogradov, A.P.Shinkarkin, A.M. Poverenniy, 2003). We decided to research pathogenetic
aspects of anemia, exchange of iron in diffuse toxic goiter. Iron deficiency (sideropenia or
hypoferremia) is one of the most common of the nutritional deficiencies. Iron is present in all cells in the human body, and has several vital functions. All cells of human body have iron. This iron has several (certain) vital function. Too little iron can negatively affect these vital functions and lead to morbidity and death.
