Abstract:
It is well known that liver cirrhosis is characterized by a pronounced fibrotic process, disruption of organ architecture, and the formation of regenerative nodules, leading to necrotizing inflammation. As a result, the internal veins of the liver and the portal vein become compressed, resulting in portal hypertension. In recent years, there has been an increase in research dedicated to the role of cytokines in the pathogenesis of liver cirrhosis and the development of disease complications.
