Abstract:
Pulmonary hypertension (PH) is a significant complication in ESRD
patients on HD, exacerbated by AV fistulas and involving dysregulation
of endothelin-1 (ET-1) and nitric oxide (NO). Aim to evaluate the role of
endothelin-1 (ET-1) and nitric oxide (NO) in the development and
progression of PH in patients on program hemodialysis and with AV
access.